Cyclin-Dependent Kinase 2 (CDK2) Over-Activation Linked to Cancer Development
CDK2 Complex Plays Critical Role in Cell Cycle Regulation
CDK2 Inhibition Emerges as Potential Cancer Treatment Strategy
A recent study has uncovered a crucial link between the over-activation of cyclin-dependent kinase 2 (CDK2) and the development of various cancers. CDK2 is a protein complex that plays a vital role in regulating the cell cycle, ensuring orderly cell division and growth.
The study revealed that the CDK2 complex is significantly over-activated in many types of cancer, including breast, lung, and colon cancer. This over-activation allows cancer cells to divide and proliferate uncontrollability, contributing to tumor growth and metastasis.
Given the crucial role of CDK2 in cancer development, researchers are investigating the potential of CDK2 inhibitors as a promising new treatment strategy. By specifically targeting and inhibiting CDK2, it may be possible to disrupt the uncontrolled cell division in cancer cells, ultimately leading to tumor regression and improved patient outcomes.
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